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dc.contributor.authorHongyu, Chai-
dc.contributor.authorPeng, Cao-
dc.date.accessioned2023-03-16T04:17:35Z-
dc.date.available2023-03-16T04:17:35Z-
dc.date.issued2023-
dc.identifier.urihttps://link.springer.com/article/10.1007/s44254-023-00011-9-
dc.identifier.urihttps://dlib.phenikaa-uni.edu.vn/handle/PNK/6918-
dc.descriptionCC BY-
dc.description.abstractThe researchers mapped the projections of AP neurons and found that excitatory and inhibitory AP neurons displayed different projection patterns. Excitatory neurons projected to many brain areas, including the parabrachial nucleus (PBN), nearby NTS, and autonomic motor nuclei, while inhibitory neurons formed connections with AP neurons themselves and partly with NTS neurons. Clusters 2 and 4 primarily projected to calcitonin gene-related peptide (CGRP) + PBN neurons, previously reported as alarm neurons [3]. These results revealed the possible circuit underlying nausea sensation.vi
dc.language.isoenvi
dc.publisherSpringervi
dc.subjectcalcium sensing receptor-
dc.subjectnausea-associated behaviors-
dc.titleTwo neural pathways for toxin-induced defensive responsesvi
dc.typeBookvi
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