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DC Field | Value | Language |
---|---|---|
dc.contributor.author | Ting, Wang | - |
dc.contributor.author | Man, Niu | - |
dc.contributor.author | Rui, Liu | - |
dc.date.accessioned | 2023-03-23T03:25:08Z | - |
dc.date.available | 2023-03-23T03:25:08Z | - |
dc.date.issued | 2023 | - |
dc.identifier.uri | https://link.springer.com/article/10.1186/s12890-023-02351-5 | - |
dc.identifier.uri | https://dlib.phenikaa-uni.edu.vn/handle/PNK/7088 | - |
dc.description | CC BY | vi |
dc.description.abstract | Anaplastic lymphoma kinase (ALK) rearrangement, which is mostly showed as fused with echinoderm microtubule-associated protein-like 4 gene (EML4), accounts 3–7% of all common mutations in non-small lung cancer (NSCLC). An intergenic region (chr2: 30,193,816), which located on upstream of the adjacent ALK gene, was never been reported as a ALK patterner before. | vi |
dc.language.iso | en | vi |
dc.publisher | Springer | vi |
dc.subject | Anaplastic lymphoma kinase | vi |
dc.subject | non-small lung cance | vi |
dc.title | A novel intergenic region (chr2: 30,193,816)-ALK fusion shows sensitivity to Alectinib in lung adenocarcinoma | vi |
dc.type | Book | vi |
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